There is evidence that links increased fat intake to the pathogenesis of IBD. Activation of Gpra, receptor for niacin and the commensal metabolite butyrate, suppresses colonic inflammation and carcinogenesis. Cite this article Liu, Y. Wu HT Regulative effect of glycine on intestinal injury and muscle protein synthesis and degradation of piglets after lipopolysaccharide challenge. The interactions between dietary nutrients and intestinal immunity are complex. Popular exclusionary diets for inflammatory bowel disease: the search for a dietary culprit. Recent studies in pigs indicate that specific dietary amino acids, in particular, arginine, glutamine, glycine, cysteine, NAC, and proline can regulate the intestinal microbial milieu and host immune system by fine-tuning inflammatory cytokine secretion and the redox status of intestinal cells, and thus exert protective effects on cells. Of note, the gut resident bacteria are capable of catabolizing polyphenols and degrading them into small fragments Footnotes Disclaimer The author is solely responsible for the findings and the content of the paper. Inflammatory bowel disease and anxiety: links, risks, and challenges faced.
Inflammatory bowel disease IBD is a chronic and relapsing inflammatory disease of the gastrointestinal tract. Although the precise etiology of IBD remains incompletely understood, accumulating evidence suggests that various environmental factors, including dietary nutrients, contribute to its pathogenesis. Dietary nutrients are known to have an impact on host physiology and diseases. The interactions between dietary nutrients and intestinal immunity are complex. Dietary nutrients directly regulate the immuno-modulatory function of gut-resident immune cells. Likewise, dietary nutrients shape the composition of the gut microbiota. Therefore, a well-balanced diet is crucial for good health. Genetic predispositions and gut dysbiosis may affect the utilization of dietary nutrients. Moreover, the metabolism of nutrients in host cells and the gut microbiota may be altered by intestinal inflammation, thereby increasing or decreasing the demand for certain nutrients necessary for the maintenance of immune and microbial homeostasis. Herein, we review the current knowledge of the role dietary nutrients play in the development and the treatment of IBD, focusing on the interplay among dietary nutrients, the gut microbiota and host immune cells.
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